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Microbiology Pathology Mnemonics

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Catalase positive organims
SPACE:
Staphylococcus aureus
Pseudomonas
Aspergillus
Candida
Enterobacter
Common cold: viral causes
"Common cold (acute infectious rhinitis, coryza) isPRIMArily caused by":
Paramyxoviruses
Rhinoviruses
Influenza viruses
Myxoviruses
Adenoviruses
E. coli: diseases caused in presence of virulence factors
DUNG:
Diarrhea
UTI
Neonatal meningitis
Gram negative sepsis·
Dung, since contract E. coli from dung-contaminated water.
Staphylococcus aureus: diseases caused
SOFT PAINS:
Skin infections
Osteomyelitis
Food poisoning
Toxic shock syndrome
Pneumonia
Acute endocarditis
Infective arthritis
Necrotizing fasciitis
Sepsis
Endotoxin features
ENDOTOXIN:
Endothelial cells/ Edema
Negative (gram- bacteria)
DIC/ Death
Outer membrane
TNF
O-antigen
X-tremely heat stable
IL-1
Nitric oxide/ Neutrophil chemotaxis
Meningitis: most common organisms
NHS:
Neisseria meningitidis
Hemophilus Influenzae
Streptococcus pneumonia
·The cause of the most
Severe meningitis isStreptococcus
.· Note: NHS is an acronym for National Health Service in several countries.
Klebsiella details
You tell the patient:
"Get UPSyou fat alcoholic":
UTI
Pneumonia
Sepsis
Fat capsule
Get up=nonmotile since no flagella
.Alcoholic=commonly seen in alcoholic and nosocomial patients.
IgA protease-producing bacteria
"Nice Strip of Ham":
Neisseria
Streptococcus pneumonia
Haemophilus influenza
E. coli: major subtypes, key point of each
"HIT by E. coli outbreak":
EnteroHemorrhagic:· HUS from Hamburgers
EnteroInvasive:·
Immune-mediated
Inflammation
EnteroToxigenic:· Traveller's diarrhea
A-B toxin producing bacteria
ABCDE
  • A - pseudomonas Aeruginosa
  • B - Bordetella pertussis
  • C - Cholera
  • D - Diptheria
  • E - E. coli (enteroinvasive)
The B subunit binds, then theA subunit ADP-ribosylates.
A and D produce A-B toxins that inhibit Elongation Factor, which is required for mRNA translation
.C and E (enteroinvasive) produce a toxin that ADP ribosylates the GTPase of the alpha subunit of GsB ADP ribosylates an activating site on Gi, thus inhibiting the inhibitor
Virchow's triad (venous thrombosis)
"VIRchow":
Vascular trauma
Increased coagulability
Reduced blood flow (stasis)
Complement: function of C3a versus C3b
C3a: Activates Acute [inflammation]
.C3b: Bonds Bacteria [to macrophages--easier digestion].
· If wish to know more than just C3:C3a, C4a, C5a activate acute.C3b, C4b bind bacteria.
Hypersensitivity reactions: Gell and Goombs nomenclature
ACID· From I to IV:
Anaphylactic type: type I
Cytotoxic type: type II
Immune complex disease: type III
Delayed hypersensitivity (cell mediated): type IV

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