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Microbiology Pathology Mnemonics

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Catalase positive organims
Staphylococcus aureus
Common cold: viral causes
"Common cold (acute infectious rhinitis, coryza) isPRIMArily caused by":
Influenza viruses
E. coli: diseases caused in presence of virulence factors
Neonatal meningitis
Gram negative sepsis·
Dung, since contract E. coli from dung-contaminated water.
Staphylococcus aureus: diseases caused
Skin infections
Food poisoning
Toxic shock syndrome
Acute endocarditis
Infective arthritis
Necrotizing fasciitis
Endotoxin features
Endothelial cells/ Edema
Negative (gram- bacteria)
DIC/ Death
Outer membrane
X-tremely heat stable
Nitric oxide/ Neutrophil chemotaxis
Meningitis: most common organisms
Neisseria meningitidis
Hemophilus Influenzae
Streptococcus pneumonia
·The cause of the most
Severe meningitis isStreptococcus
.· Note: NHS is an acronym for National Health Service in several countries.
Klebsiella details
You tell the patient:
"Get UPSyou fat alcoholic":
Fat capsule
Get up=nonmotile since no flagella
.Alcoholic=commonly seen in alcoholic and nosocomial patients.
IgA protease-producing bacteria
"Nice Strip of Ham":
Streptococcus pneumonia
Haemophilus influenza
E. coli: major subtypes, key point of each
"HIT by E. coli outbreak":
EnteroHemorrhagic:· HUS from Hamburgers
EnteroToxigenic:· Traveller's diarrhea
A-B toxin producing bacteria
  • A - pseudomonas Aeruginosa
  • B - Bordetella pertussis
  • C - Cholera
  • D - Diptheria
  • E - E. coli (enteroinvasive)
The B subunit binds, then theA subunit ADP-ribosylates.
A and D produce A-B toxins that inhibit Elongation Factor, which is required for mRNA translation
.C and E (enteroinvasive) produce a toxin that ADP ribosylates the GTPase of the alpha subunit of GsB ADP ribosylates an activating site on Gi, thus inhibiting the inhibitor
Virchow's triad (venous thrombosis)
Vascular trauma
Increased coagulability
Reduced blood flow (stasis)
Complement: function of C3a versus C3b
C3a: Activates Acute [inflammation]
.C3b: Bonds Bacteria [to macrophages--easier digestion].
· If wish to know more than just C3:C3a, C4a, C5a activate acute.C3b, C4b bind bacteria.
Hypersensitivity reactions: Gell and Goombs nomenclature
ACID· From I to IV:
Anaphylactic type: type I
Cytotoxic type: type II
Immune complex disease: type III
Delayed hypersensitivity (cell mediated): type IV


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